Liver dystrophy

Liver dystrophy

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FATTY LIVER DYSTROPHY (arrows indicate large drops of fat).

Liver dystrophy(dystrophia hipatis) - parenchymal degeneration of the liver, characterized by excessive accumulation of glycogen in hepatocytes and a significant increase in organ.

D.P.acute, hepatic coma- common necrosis (tissue death) of the liver caused by exposure to toxic substances of both exogenous and endogenous origin. The most common cause of AD is acute in children is acute viral hepatitis (see Hepatitis viral), as well as other hepatitis. Rarely develops with alimentary dystrophy and preanaphylactic shock. Nephrosis of the parenchyma of the entire hepatic lobule is preserved morphologically with preservation of the stroma of the liver.

PATHOGENESIS is associated with funkts. liver failure, which leads to accumulation in the body of toxic. metabolic products (ammonia, phenols, etc.).

A CLINICAL PICTURE develops gradually, less often at lightning speed (within a few hours). Characterized by: weakness, drowsiness, psychomotor agitation, increasing jaundice,prog-diminished liver size, hemorrhagic syndrome (accompanied by bleeding and hemorrhage), Cus-Smalya respiration, “hepatic smell” from the mouth, the appearance of pathological reflexes, convulsions. Sometimes hepatorenal syndrome (combination of jaundice, impaired blood coagulation, signs of hepatic reflexes, and spasms. renal failure). TREATMENT D. acute neural syndrome: detoxification, hepatoprotective (hepatic-but-protective), hemostatic and vitamin therapy, the introduction of proteolysis inhibitors needle-cockort oids in large doses, hemosorbium, hyperbaric oxygenation. The treatment is carried out in the intensive care unit.

D.P. fatty, fatty hepatosis, liver obesity- a disease characterized by intracellular accumulation of lipids in hepatocytes (secretory cell of the liver parenchyma). PATHOGENESIS is based on lipid metabolism disorders, due to which an imbalance occurs between the flow of lipids into the liver and their metabolism in the liver, the formation and release of lipoproteins from the liver. In children it develops as a result of an excess intake of fats from food, with metabolic diseases (Kushin-ga syndrome,diabetes mellitus, etc.) and fermentopathies - the absence or disruption of enzyme activity: glycogenosis, cholesterol nemia (elevated cholesterol in the blood serum), Wolman's disease (hereditary disease, intracellular accumulation of cholesterol esters and triglycerides), etc.

CLINICAL PICTURE. Progressive hepatic insufficiency with dysplasia, jaundice, hemorrhagic diathesis, hepatomegaly (significant enlargement of the liver) are characteristic of D. adiposectomy, slightly altered func. samples of the liver. Sometimes it is possible to develop chronic: hepatitis. DIAGNOSIS is established on the basis of anamnestic (see Anamnesis) and clinical biochem. data. A crucial role in the diagnosis of D. of adipose fat is played by puncture biopsy of the liver.

TREATMENT. A diet that contains high-quality proteins and vitamins in an optimum way, and an insignificant amount of fats, lipotropic agents (choline-chloride, lipamide, etc.), liver hydrolysates (prohepar, etc.), vitamin B12and etc.

PREVENTION: diet, nutrition, treatment, primary. diseases.

LIVER DISTROPHY:


a - carbohydrate dystrophy (arrows indicate vacuolation of hepatocyte nuclei);


b - in case of bridge necrosis (arrows indicate the area of ​​the bridge necrosis connecting the two portal tracts);


c - in case of cirrhosis (arrows indicate sharply dilated lymphatic vessels among the growths of the connective tissue);


d - intracellular regeneration (a giant multinuclear hepatocyte is visible in the center).



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  • Liver dystrophy

    Liver dystrophy

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